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Thyroid Scale Matrix

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Below is the Thyroid Scale Interpretation Matrix. Remember that lab values are noted in relation to optimal and not standard lab normal/abnormal. This tool is extremely useful on its own, but including information from the Metabolic Scorecard™ and/or Metabolic Temperature Graph™ only yields more accurate information. Some relevant feedback from these tools where added to the below matrix as well. Note that taking thyroid hormone replacement or some herbal or iodine supplementation may change the interpretation of the matrix.

Thyroid Scale™ Interpretation Matrix
State of Health TSH T4 T3 Temperature Pattern Commentary
Healthy Optimal Optimal Optimal 98.6, stable
Adrenal Fatigue Low Low Low Low, average is typically 97.8 or lower. Very unstable Symptoms: Predominate in adrenal column. Often confused with hypothyroidism because of low T4 and T3. Some doctors mistakenly interpret the low TSH here to mean pituitary trouble.
Estrogen Dominance (ED) Low Low Low Low, average is typically 97.8 or lower and unstable Is the same as adrenal fatigue in that both are related to each other. In adrenal fatigue, the adrenals often sequester the progesterone to help make cortisol. The drop in progesterone creates the progesterone / estrogen imbalance called ED.
Hypothyroidism due to low thyroid function as a primary cause, e.g., Surgical removal of thyroid with insufficient replacement of T4 High Low Low but to the right of T4 Low and very stable Note there is high conversion of T4 to T3. There is a high demand for T4/T3 (high TSH) and the body is extracting as much T3 out of the T4 as it can.
Hypothyroidism due to low pituitary function Low Low Low but to the right of T4 Low and very stable Looks just like primary hypothyroidism but TSH is low. We know there is demand because of high conversion of T4 to T3 but the TSH doesn’t rise to help T4 production.
Late Hashimoto’s Thyroiditis or Hypothyroid and Adrenal fatigue Optimal to high Low Low and mildly to the Right of T4 Low and unstable The most common presentation of disease. Similar to adrenal fatigue but symptoms are predominately in the mixed column.
Early Hashimoto’s Thyroiditis Very low High High but to the left of T4 Can range from below 98.6 to slightly above. The body can slow down metabolism (step on the brakes) by shifting conversion of T4 toward RT3 and away from T3. Thus we see T3 is to the left of T4.
Grave's Disease Very low Very high Very high and to the right of T4 Tends to be above 98.6 and stable in early phase. Later, drops below 98.6 and becomes unstable. T3 to the right of T4 (i.e., high conversion of T4 to T3) is like a car that’s speeding out of control and the driver steps on the accelerator. This is typical for Grave’s Disease.
Poorly effective thyroid hormone Mildly high High High and to the right of T4 Low and moderately stable Can be due to nutrient deficiency, viral damage to mitochondria, toxic burden, or poor receptor site (to thyroid hormone) sensitivity.
Chronic infection Optimal to mildly high Optimal to mildly high Optimal to mildly high Mildly above 98.6 Source of infection may be elsewhere.
On thyroid support that contains T3:
  • Dessicated thyroid such as Armour thyroid
  • T4/T3 mixture such as Thyrolar
  • Slow release T3 (compounded)
  • Pure fast releaseT-3 such as Cytomel
Optimal if dose is proper.

High if dose is too low.

Low if does is too high.
If the TSH is optimal, the T4 will be low. If the TSH is optimal, the T3 is high Often unstable. Tends to be less unstable with Armour and most unstable with fast release T3 (Listed in the first column in order of increasing destabilizing effect on the temperature pattern). The body seems to do better with a steady state of energy. T4 acts slowly but T3 is rapid to come and leave. Therefore, to maintain a steadier blood level of T3, it is best taken in divided doses. Splitting the daily dose into 2 or 3 portions stresses the adrenals less than taking the entire dose once daily and therefore leads to more stable temperatures.
Above matrix is to be used with Dr. Rind's Thyroid Scale™


1. Hashimoto’s Thyroiditis is a common autoimmune condition in which one develops an allergy to one’s own thyroid gland. In the early phase when there is destruction of thyroid gland and spillage of thyroid hormone (T4), there is a hyperthyroid effect. In an effort to lower the T4 level in the blood, the pituitary gland decreases the amount of TSH it secretes producing a low TSH. The hyper-metabolic state that occurs usually stresses the adrenal glands and causes adrenal fatigue. When enough destruction has occurred and the thyroid gland can make only a small amount of T4, one goes into a hypothyroid phase. Now one has hypothyroidism and adrenal fatigue. Autoimmune antibodies, Anti Thyroglobulin Antibodies (ATA) and Thyroid PerOxidase Antibodies (TPO), are almost always present on blood testing. The body can eventually counter the hyper-metabolic state by reducing the conversion of T4 to T3 (and increasing T4 to RT3 conversion). Thus metabolically, this is like stepping on the brakes in a car that’s going too fast.

2. Grave’s Disease is an autoimmune disease in which an antibody is produced that mimics TSH. It signals the thyroid gland to make T4. As the T4 level rises, the pituitary tries to reduce the T4 level by reducing TSH levels and we get a low TSH. Typically we find elevation of Thyroid Stimulating Immuneglobulin or TSI. Most labs consider a level of 130 or higher as evidence of Grave’s Disease. In reality, we often see the signs of hyperthyroidism begin to appear in a subtle way at a level of 90. At 110 the symptoms are easier to see. By the time we get to 130 the symptoms are usually severe. Unlike Hashimoto’s Thyroiditis, in Grave’s disease the T4 goes into high conversion to T3. This is like driving a car too fast and stepping on the accelerator. This is extremely stressful to the adrenals.

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